The goal of our publication is to communicate the pictures of spontaneous findings occurring in beagles widely. home window Fig. 53. Abdomen: Infections by in the gastric mucosa. infections occurs in the lumen from the pyloric gland without irritation occasionally. Many beagles possess in their abdomen. Fig. 54. Abdomen: Mineralization in the lamina propria Open up in another home window Fig. 54. Abdomen: Rabbit polyclonal to ACCS Mineralization in the lamina propria. Focal mineralization is certainly common in the lamina propria. Fig. RAD001 tyrosianse inhibitor 55. Duodenum: Hyperplasia from the lymphoid tissues Open up in another home window Fig. 55. Duodenum: Hyperplasia from the lymphoid tissues. Hyperplasia from the lymphoid tissues occurs in not merely the duodenum but other intestinal tracts occasionally. Advancement of the intestinal lymphoid tissues varies among individual animals. Fig. 56. RAD001 tyrosianse inhibitor Duodenum: Dilatation of the crypt Open in a separate window Fig. 56. Duodenum: Dilatation of the crypt. Dilatation of the crypt filled with/without cell debris or inflammatory cell infiltration is frequently seen. The change most frequently occurs in the duodenum but also occurs in the other intestines. Fig. RAD001 tyrosianse inhibitor 57. Duodenum: Ectopic pancreatic tissue Open in a separate window Fig. 57. Duodenum: Ectopic pancreatic tissue. Ectopic pancreatic tissue is frequently seen in the submucosa near the major duodenal papilla. Fig. 58. Jejunum: Ectopic fundic gland Open in a separate window Fig. 58. Jejunum: Ectopic fundic gland. An ectopic fundic gland is rarely seen in the jejunum. The ectopic fundic gland is composed of normal fundic tissue. Fig. 59. Cecum: Granular cell tumor Open in a separate window Fig. 59. Cecum: Granular cell tumor. Large polygonal eosinophilic cells infiltrate into the lamina propria and submucosa of the cecum. The tumor cells have fine granular cytoplasm. This tumor is rare in beagles. Fig. 60. Salivary gland: Focal inflammatory cell infiltration Open in a separate window Fig. 60. Salivary gland: Focal inflammatory cell infiltration. Focal lymphocytic infiltration, occasionally accompanied by lymph follicle formation, is common around the ducts or acini. Fig. 61. Salivary gland: Focal fibrosis Open in a separate window Fig. 61. RAD001 tyrosianse inhibitor Salivary gland: Focal fibrosis. Focal fibrosis with atrophy and loss of the acini is frequently seen, especially in the parotid RAD001 tyrosianse inhibitor gland. Fig. 62. Salivary gland: Salivary calculus Open in a separate window Fig. 62. Salivary gland: Salivary calculus. A small mineralized calculus occurs occasionally in a duct of a relatively large caliber. Fig. 63. Salivary gland: Increase in the mucous gland Open in a separate window Fig. 63. Salivary gland: Increase in the mucous gland. The mucous epithelium increases occasionally in the parotid gland, though the parotid gland of the beagles is composed mainly of a serous epithelium. Fig. 64. Liver: Accumulation of glycogen in hepatocytes Open in a separate window Fig. 64. Liver: Accumulation of glycogen in hepatocytes. The change is common in beagles and characterized by a clear appearance of hepatic cytoplasm after formalin fixation. The quantity of glycogen in cytoplasm varies among individuals under normal conditions. Fig. 65. Liver: Microgranuloma Open in a separate window Fig. 65. Liver: Microgranuloma. Microgranuloma is accumulation of inflammatory cells, mainly macrophages, lymphocytes and a small number of neutrophils, and may be associated with minute necrosis of hepatocytes. It is found frequently. Fig. 66. Liver: Pigment deposition in hepatocytes Open in a separate window Fig. 66. Liver: Pigment deposition in hepatocytes. Lipofuscin pigments are common in centrilobular hepatocytes.